Fig. 5From: Protective role of boron on hepatotoxicity and oxidative stress induced by trichloroacetic acidPossible signaling pathway involved in the protective role of B on the antioxidant, anti-inflammatory, or cell apoptosis. B may reduce oxidative damage and inflammatory responses induced by TCA. The activation of p38 MAPK, total p38 MAPK in the nucleus decreases, and p21 downregulation causing HepG2 cells apoptosis might be related with B’s protective roleBack to article page