Fish | NPs type | Concentration selection method | Duration | Organs analyzed | Histological study | Bioaccumulation | Reference |
---|---|---|---|---|---|---|---|
Common carp fish | AgNPs | 0.04, 0.08 mg/L | 96 h | Gills, liver, and intestine | Epithelial lifting, telangiectasia in secondary lamellae, and epithelial necrosis | Mostly in the liver and intestine | [27] |
Zebrafish, rainbow trout fish | CuNPs | 1 mg/L | 96 h | Gills | Damage of gill filaments | N/A | [28] |
Epinephelus coioides | CuNPs | 100 μg/L | 25 days | Gills, intestine, liver, and muscles | Cell apoptosis in gills, liver | Liver, gills, intestine, and muscles | [29] |
Common carp fish | AgNPs | 0.75 mg/L | 14 days | Gills | Telangiectasia and epithelial cell hyperplasia in fish gills | N/A | [5] |
Common carp fish | ZnONPs | 50 mg/L | 30 days | Gills, liver intestine, and brain | Irregular cell outline, abnormal pyknotic nuclei, shrinkage of cell | Mostly accumulated in the liver and gills | [30] |
Common carp fish | CuNPs | N/A | 21 days | Gills, intestine, and liver | N/A | Mostly accumulated in the liver and intestine | [31] |
Common carp fish | B-AgNPs | 0.03, 0.06, 0.09 mg/L | 20 days | Gills, intestine, muscles, liver | Histological alteration mostly occurred at the highest concentration of B-AgNPs in gills and intestine | Widely accumulated in the liver, followed by the intestine, gills, and muscle | Our study |